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Burton J. Kallman

Publications and source records attributed to Burton J. Kallman.

3 recordsLinked to original sources

Reductive dechlorination of DDT to DDD by yeast

Labeled DDD [ 1,1-dichlor-o-2,2-bis(p-chlorophenyl)-ethane] was formed from C 14 -labeled DDT in the presence of yeast. The formation of DDD from DDE [1,1-dichloro-2,2-bis (p-chlorophenyl)-ethylene] was not observed, indicating that a reductive dechlorination of DDT occurs.

Science

Insecticides: effects on cutthroat trout of repeated exposure to DDT

Cutthroat trout were periodically exposed to p, pp-DDT, in acetone solution or in the food. Excessive mortality occurred only in lots treated with high concentrations of DDT, probably as a result of decreased resistance to nonspecific stressors. Surviving fish in these lots were significantly larger than those in the control lot, or in the lots treated with low concentrations of DDT. The number and volume of eggs produced was not reduced by DDT, but mortality among sac fry appeared to be highest in the lots treated with high concentrations. The data suggest that the sublethal concentrations of DDT ordinarily encountered in the environment are unlikely to damage a fishery.

Science

Distribution and detoxication of toxaphene in Clayton Lake, New Mexico

The fate of toxaphene, applied in three treatments at a total calculated concentration of 0.05 p.p.m. to Clayton Lake, New Mexico, was followed over a 1.5-year period. A detailed description of the chromatographic method of analysis is given. Water concentrations of toxaphene were higher in leeshore samples than in windward samples for 2 weeks after the application; toxaphene levels then appeared to reach a constant value of about 0.001 p.p.m. for at least an additional 250 days. Total body concentrations of toxaphene were determined in trout and bullheads present in the lake during the poisoning and in trout placed in the lake in live-cars subsequently. Trout were more susceptible to toxaphene and accumulated lower body levels than bullheads. Bullheads which showed symptoms of toxaphene poisoning when collected had higher levels than did normal-appearing individuals. No difference in levels was observed in live-car trout collected dead as compared to survivors. Aquatic vegetation accumulated high concentrations of toxaphene; low concentrations were found in some sediment samples. The significance of these findings is discussed.

New Mexico